WHEN Sabine Boghos still hadn't started her periods by the age of 16, docs thought that she might just be underweight. But it wasn't long before she received the devastating news that she had never developed a womb and a third of her vaginal canal was missing. The year-old has MRKH syndrome - a rare condition which leaves the reproductive system underdeveloped.
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What is vaginal agenesis?
They occur while she is growing in her mother's womb. Female reproductive organs include the vagina , ovaries, uterus, and cervix. A baby starts to develop its reproductive organs between weeks 4 and 5 of pregnancy. This continues until the 20th week of pregnancy. The development is a complex process. Many things can affect this process. How severe your baby's problem is depends on when the interruption occurred.
How we care for vaginal agenesis
That may be because, as with many things related to the vagina, much is misunderstood or left up to myth and urban legend. The average vaginal canal is 3 to 6 inches long, or about the length of your hand. For example, if you become sexually aroused, your vaginal canal grows longer. This allows your cervix and uterus to lift up and away from the canal so that penetration will be more comfortable. The vaginal canal undergoes many changes, and no two are same. All of this is normal. And unless you feel any pain or discomfort , everything is likely fine. It could be caused by an underlying condition that makes penetration uncomfortable. You may think uncomfortable penetration is only a side effect from sex with someone who has a penis. For example, you may experience discomfort when trying to insert a tampon, menstrual cup, or sex toy.
Vaginal atresia is a condition in which the vagina is abnormally closed or absent. The main causes can either be complete vaginal hypoplasia , or a vaginal obstruction, often caused by an imperforate hymen or, less commonly, a transverse vaginal septum. This condition does not usually occur by itself within an individual, but coupled with other developmental disorders within the female. The exact mechanism for vaginal atresia is not well known, as specific molecular mechanisms which lead to the closing or absence of the vagina are unclear. There are various pathways that may support or restrict regular vaginal development. Research has shown that changing factors may also include paracrine and autocrine signals and changes in the basis of developing organs. Specific patterns of genetic transmission have not been identified for this condition. The abnormal development of the vagina results in an incomplete unit low, mid, high transverse septum , failure of epithelium degeneration imperforate hymen , and vaginal atresia.